The
parietal cortex and episodic memory: An attentional account.
(2008) Neural correlates of social target value in macaque
parietal cortex. Curr.
One study found that during verbal working memory, binge drinking young adults had greater activation in the
parietal cortex (pre-supplementary motor area) than control subjects.
Based on an ICA of resting-state fMRI data from all subjects, increased activation was found in the medial prefrontal area, precuneus, and bilateral
parietal cortex. The coordinates and Z scores for the DMN areas are described in Table 2.
Epileptic events are usually associated with motor findings; however, lesions involving the
parietal cortex can present as focal sensory seizures, which often present a diagnostic dilemma as the symptoms may be tingling, numbness, or dysesthesias [1].
Cortical processing after visual stimulus entails the participation of multiple and widespread brain areas, such as the
parietal cortex [3] and frontal cortices [4, 5].
The current work shows for the first time that even a very short single train of rTMS (intermittent TBS (iTBS)) applied over the left posterior
parietal cortex (i.e., an area shown to be activated during the Stroop task performance [5]) may enhance cognitive speed in HYS.
Surface-based analysis which provides insight into the cortical morphological variations has identified shape differences between MCI converters and nonconverters in the inferior
parietal cortex and both lateral and medial temporal cortices, especially in the entorhinal cortex [11, 13, 14].
Attention, motor control and motor imagery in schizophrenia: Implications for the role of the
parietal cortex. Schizophr Res.
Electrocorticography with a noncephalic point of reference was used to detect brain activity: one electrode was positioned in the frontal cortex (AP = -2; L = +2.5) and one in the
parietal cortex (AP = -6; L = +2.5) according to the atlas coordinates of Paxinos and Watson and a ground electrode was placed on the nasal bone.
Regarding the physiological interpretation of the regional hypoperfusion consistently found in the PCC, precuneus and/or lateral
parietal cortex, a debate exists as to whether this is a cause or consequence of the disease [33].
